Pathophysiology Frequency History Physical Causes Contacts

Physical

The physical examination of patients who have not yet manifested neurologic dysfunction may be misleading. The occipital and nuchal pain associated with VAD mimics musculoskeletal pain and often is attributed to the mechanical strain that precipitated the dissection.


1) Depending upon which areas of the brain stem or cerebellum are experiencing ischemia, the following signs may be present:

  • Limb or truncal ataxia
  • Nystagmus
  • Ipsilateral Horner syndrome in as many as one third of patients with VAD (ie, impairment of descending sympathetic tract)
  • Ipsilateral hypogeusia or ageusia (ie, diminished or absent sense of taste)
  • Ipsilateral impairment of fine touch and proprioception
  • Contralateral impairment of pain and thermal sensation in the extremities (ie, spinothalamic tract) \
  • Lateral medullary syndrom

2) Cerebellar findings may include the following:

  • Nystagmus
  • Medial medullary syndrome
  • Tongue deviation to the side of the lesion (impairment of CN XII)
  • Contralateral hemiparesis
  • Ipsilateral impairment of fine touch and proprioception (nucleus gracilis)
  • Internuclear ophthalmoplegia (lesion of the medial longitudinal fasciculus)

 

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